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Because FOXO4-DRIs mechanism depends on disrupting the FOXO4p53 interaction, and because this interaction is only operative in cells with elevated FOXO4 expression, the compound preferentially targets the senescent cell population leaving FOXO4-low non-senescent cells largely unaffected

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Research has confirmed that the pro-fibrotic factor Wnt-1-induced signaling protein 1 (WISP-1) may mediate renal fibrosis by enhancing autophagy mediated by TGF-1 (195, 196)

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