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PMID 21296191

(2004) Proceedings of the National Academy of Sciences (PNAS) , 101(39): 1490714912 Foundational study identifying the EPO/-common receptor heteromer (IRR) as the mediator of EPOs tissue-protective effects, distinct from the EPOR homodimer responsible for erythropoiesis

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The consequent accumulation of BACH1 was shown to enhance critical processes in vascular regeneration, namely, the proliferation and tube-forming capacity of vascular endothelial cells

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