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It was previously reported that targeted FUS-induced BBB opening facilitates CNS penetration of systemically administered anti-A antibodies to targeted brain regions in the TgCRND8 and APP23 mouse model of AD, therefore reducing plaque load 5 , therefore confirms the feasibility of this strategy in promoting behavioral and memory recovery in a transgenic animal model 6,7

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EDC/NHS chemistry was used to graft functional groups (alanine and GSH), couple therapeutic molecules (ibuprofen and dopamine) on PLG backbone and label the co-polypeptides with R6G (Fig

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However, long-term oral NAC administration is associated with higher risk of RA [141], and potential clinical application of NAC requires further study

Because of this, the new treatment didnt quite meet the studys goal of proving it works just as well as (or better than) Zepbound