Transcriptomic analysis reveals regulatory genes effects from ECM1-modified MenSCs To gain insight into the potential mechanisms by which ECM1-modified MenSCs attenuate liver fibrosis independently of the ECM1LRP1 signaling axis, transcriptomics was performed to compare changes in transcript levels
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RBCs use the cofactor S-adenosyl-homocysteine (SAM) and Protein-L-isoaspartate O-methyltransferase (PCMT1) system, to repair proteins damaged by oxidative stress 49
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